Hip

Hip Osteoarthritis

Hip osteoarthritis is the most common cause of chronic hip pain in adults over 50. It is also one of the leading reasons people in the Washington, D.C. metro area seek orthopedic care. At Maryland Orthopedic Specialists, our adult reconstruction team treats every stage of hip arthritis, from early cartilage loss managed with physical therapy and injections to end-stage disease requiring total hip arthroplasty. Our goal is to relieve pain, restore function, and keep you active as long as possible with the least invasive approach that works for you.

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In-network with most major insurance plans. Same-day appointments available for acute injuries.

What is hip osteoarthritis?

Hip osteoarthritis (OA) is a degenerative joint disease characterized by the progressive breakdown of articular cartilage, the smooth, low-friction lining that covers the femoral head and acetabulum. As cartilage thins and erodes, bone rubs on bone, triggering pain, inflammation, reactive bone spur (osteophyte) formation, and eventual joint-space narrowing visible on X-ray.

Hip osteoarthritis (OA) is a degenerative joint disease characterized by the progressive breakdown of articular cartilage, the smooth, low-friction lining that covers the femoral head and acetabulum. As cartilage thins and erodes, bone rubs on bone, triggering pain, inflammation, reactive bone spur (osteophyte) formation, and eventual joint-space narrowing visible on X-ray.

Key facts:

  • Affects an estimated 10–25% of adults over age 60
  • Primary OA develops without a clear underlying cause; secondary OA results from prior hip conditions such as femoroacetabular impingement (FAI), hip dysplasia, childhood Legg-Calvé-Perthes disease, avascular necrosis, or prior trauma
  • Both hips may be affected, though often asymmetrically

The C-Sign: Patients frequently describe groin pain by cupping their hand in a "C" shape over the front of the hip, with fingers toward the groin and thumb toward the buttock. This is a classic clinical indicator of intra-articular hip pathology, including OA.

Gait changes: Advanced OA commonly produces an antalgic gait (a shortened stance phase on the affected side to minimize pain) or a Trendelenburg gait if abductor muscles weaken secondarily.

Symptoms — do you recognize these?

  • Deep groin pain, often described as aching or throbbing
  • Pain with weight-bearing activities: walking, stair climbing, rising from a chair
  • Morning stiffness lasting less than 30 minutes (longer stiffness suggests inflammatory arthritis)
  • Reduced hip range of motion — difficulty putting on shoes and socks, getting in and out of a car
  • Grinding or catching sensation (crepitus) in the joint
  • Referred pain to the thigh, buttock, or knee
  • Positive FABER test (Flexion, ABduction, External Rotation) and FADIR test (Flexion, ADduction, Internal Rotation) on physical examination — internal rotation is typically the first motion lost in hip OA

Call us at (301) 515-0900. Same-day appointments available at our Bethesda and Germantown locations.

How we diagnose it

Physical examination remains central. Your provider will assess range of motion, perform provocative tests (FABER, FADIR, log roll), evaluate gait, and screen for referred pain from the lumbar spine.

Weight-bearing X-rays are the primary imaging tool. Standing AP pelvis and lateral hip views reveal:

  • Joint-space narrowing (normal joint space: 3–5 mm; concerning if < 2 mm)
  • Subchondral sclerosis and cysts
  • Osteophyte formation at joint margins

MRI may be ordered when symptoms are disproportionate to X-ray findings, when a labral tear or chondral defect is suspected alongside early arthritis, or to evaluate AVN.

Diagnostic injection: An image-guided intra-articular injection of local anesthetic can confirm the hip as the pain generator, particularly when lumbar spine pathology co-exists.

Laboratory tests (ESR, CRP, CBC, uric acid) help rule out inflammatory or crystalline arthritis if the clinical presentation warrants.

Treatment options

Treatment follows a structured ladder based on symptom severity, functional limitation, and radiographic stage.

Non-Operative Treatments

Physical therapy is first-line and among the most effective interventions for mild-to-moderate hip OA. A targeted program addresses hip flexor and external rotator flexibility, hip abductor and core strengthening, and gait retraining. Aquatic therapy reduces joint load while maintaining cardiovascular conditioning. Activity modification (reducing high-impact activities (running, jumping) in favor of low-impact alternatives (cycling, swimming, elliptical)) decreases mechanical joint stress and slows symptom progression. Weight management: Each pound of body weight lost reduces hip joint loading by 3–6 pounds per step. Even a 10% reduction in body weight produces measurable symptom improvement. Oral medications: NSAIDs (ibuprofen, naproxen, celecoxib) are effective for pain and inflammation. Acetaminophen and topical diclofenac are alternatives for patients who cannot tolerate oral NSAIDs. Corticosteroid injection: Ultrasound- or fluoroscopy-guided intra-articular corticosteroid injection provides short-term pain relief (typically 4–12 weeks) and is most useful as a bridge to physical therapy or surgery. Platelet-Rich Plasma (PRP): PRP injections concentrate autologous growth factors that may modulate inflammation and support cartilage health. Emerging evidence supports their use in mild-to-moderate hip OA, with effects that may outlast corticosteroid. Viscosupplementation: Hyaluronic acid injections aim to restore joint lubrication and reduce friction; evidence in the hip is more limited than in the knee, and insurance coverage varies.

Surgical Procedure

Total Hip Arthroplasty (Hip Replacement)

Complete hip joint replacement removing the arthritic femoral head and acetabulum and replacing them with metal, ceramic, and polyethylene components. Anterior approach technique is available for eligible patients, preserving more muscle tissue.

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Recovery & rehabilitation

Recovery depends on treatment stage:

  • Physical therapy program: 6–12 weeks to functional improvement; ongoing home program
  • Corticosteroid/PRP injection: Relief typically within 1–2 weeks; activity may resume immediately
  • Total hip arthroplasty: Walking with a cane or walker within 1–2 days; most patients return to light activity in 4–6 weeks; full recovery and return to sport at 3–6 months

Frequently Asked Questions

Can hip arthritis be reversed?
Cartilage loss cannot currently be reversed, but symptoms can be effectively managed and progression slowed. Maintaining a healthy weight, staying active with low-impact exercise, and following a structured PT program are the most impactful modifiable factors.
How do I know if it's my hip or my lower back causing my groin pain?
Hip arthritis typically produces groin pain that worsens with weight-bearing and hip rotation. Back pain more commonly radiates down the leg below the knee and varies with spinal position. A diagnostic hip injection can clarify the source when both are suspected.
Is there a best age for hip replacement?
Modern implants are highly durable (90%+ at 20 years) and age alone is rarely a limiting factor. The decision is based on symptom severity, functional limitation, and failure of conservative care, not a specific age threshold.
What is the difference between PRP and a cortisone shot?
Corticosteroid injections reduce inflammation rapidly but do not address underlying cartilage changes. PRP delivers concentrated growth factors that may have regenerative effects. Many patients benefit from cortisone first for acute flares, and PRP for longer-term management.
How long after total hip replacement can I expect to be back to normal daily activities?
Most patients walk with minimal assistance within one to two days of total hip replacement and return to light daily activities (including driving, household tasks, and short walks) within four to six weeks. By three months, the majority of patients report significant pain relief and improved function compared to before surgery. Full recovery, including optimal strength and endurance, continues to improve for six to twelve months as surrounding muscles strengthen. Your MOS team will provide specific activity precautions and a rehabilitation plan to ensure a safe, efficient recovery.

Meet the specialists

Brian McCormick, MD

Brian McCormick, MD

Adult Reconstruction · Hip and Knee Replacement

Meet Dr. McCormick
John J. Christoforetti, MD

John J. Christoforetti, MD

Orthopedic Surgery · Sports Medicine · Hip, Knee & Shoulder Arthroscopy · Shoulder Replacement

Meet Dr. Christoforetti
Medically reviewed by Brian McCormick, MD
Last reviewed May 1, 2026

References

  1. Glyn-Jones S, Palmer AJ, Agricola R, et al. Osteoarthritis. Lancet. 2015;386(9991):376–387. https://doi.org/10.1016/S0140-6736(14)60802-3
  2. Arden NK, Perry TA, Bannuru RR, et al. Non-surgical management of hip osteoarthritis. BMJ. 2021;374:n1682. https://doi.org/10.1136/bmj.n1682
  3. Mow VC, Huiskes R. Basic Orthopaedic Biomechanics and Mechano-Biology. 3rd ed. Lippincott Williams & Wilkins; 2005. [Foundational reference for cartilage biomechanics]
  4. Haddad FS, Konan S, Tahmassebi J. A prospective randomised controlled trial of total hip arthroplasty versus resurfacing arthroplasty in the treatment of young patients with arthritis of the hip joint. Bone Joint J. 2015;97-B(11):1440–1450. https://doi.org/10.1302/0301-620X.97B11.36515
  5. Bannuru RR, Osani MC, Vaysbrot EE, et al. OARSI guidelines for the non-surgical management of knee, hip, and polyarticular osteoarthritis. Osteoarthritis Cartilage. 2019;27(11):1578–1589. https://doi.org/10.1016/j.joca.2019.06.011
  6. American Academy of Orthopaedic Surgeons. Hip Osteoarthritis. OrthoInfo. https://orthoinfo.aaos.org/en/diseases--conditions/arthritis-of-the-hip/
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