Podiatry (Foot & Ankle Surgery)Foot & AnkleSurgery Center

Posterior Tibial Tendon Reconstruction (Flatfoot Correction)

Dr. Gary Feldman, DPM, performs posterior tibial tendon reconstruction using a staged approach tailored to the severity of flatfoot deformity, combining FDL tendon transfer with calcaneal osteotomy, lateral column lengthening, or subtalar fusion as dictated by deformity stage.

Duration: 90–150 minutesAnesthesia: Regional or general

What is posterior tibial tendon reconstruction (flatfoot correction)?

Posterior tibial tendon reconstruction corrects adult acquired flatfoot caused by a failing posterior tibial tendon. Surgery combines a tendon transfer (flexor digitorum longus) to replace the weakened tendon with bone realignment procedures — typically a calcaneal osteotomy and sometimes lateral column lengthening — to restore the foot's arch and correct malalignment.

Why this approach — at MOS

Pre-operative planning for flatfoot reconstruction is detailed work. I obtain weight-bearing AP and lateral foot X-rays, a hindfoot alignment X-ray, and MRI. I measure the talo-first metatarsal angle (Meary's angle), calcaneal pitch, and hindfoot valgus angle to determine which components of the reconstruction are needed and in what magnitude of correction.

My standard Stage II reconstruction combines FDL transfer with medializing calcaneal osteotomy. I add lateral column lengthening when forefoot abduction is significant on weight-bearing X-ray — the threshold I use is an abduction angle that leaves significant "too many toes" sign that the MCO alone will not correct. I repair the spring ligament when it is visibly stretched or torn, because spring ligament competence is critical to maintaining the corrected talar head position after surgery.

For patients presenting from Bethesda and surrounding communities with Stage II PTTD who have had prior orthotics and PT without adequate relief, reconstruction is highly effective. Published outcomes show excellent correction of deformity and high patient satisfaction at 5–10 years. The recovery is long, but the functional improvement — including return of single-leg heel rise — justifies the investment.

I am direct with patients about what this reconstruction requires: two to three incisions, a long non-weight-bearing period for the calcaneal osteotomy to heal, and a dedicated physical therapy commitment. Patients who understand what they are getting into before surgery recover better than those who are surprised by the recovery demands.

Who is a candidate?

Indications

  • Stage II adult acquired flatfoot: flexible hindfoot valgus, partial arch collapse, failure of non-operative management
  • Persistent medial ankle pain and arch pain despite 3–6 months of orthotics and physical therapy
  • Inability to perform a single-leg heel rise on the affected side
  • Progressive deformity documented on serial weight-bearing X-rays (increasing talo-first metatarsal angle, increasing calcaneal pitch loss)
  • MRI confirming posterior tibial tendon degeneration, elongation, or partial tear

Contraindications

  • Stage I PTTD — tenosynovitis without significant deformity responds well to non-operative care; surgery is premature
  • Stage III rigid deformity in patients who prefer conservative management — surgical risk/benefit must be carefully discussed
  • Peripheral vascular disease insufficient for healing of medial and lateral incisions
  • Poorly controlled diabetes or immunosuppression compromising healing

Conservative Treatment First

Stage I and early Stage II PTTD are managed non-operatively. The foundation is a rigid or semi-rigid custom orthotic with a medial arch support and hindfoot posting to correct heel valgus. This orthotic must be worn in all footwear — patients who use orthotics only some of the time have worse outcomes. A University of California Biomechanics Laboratory (UCBL) custom insert or an ankle-foot orthosis (AFO) provides more robust support for more advanced non-operative cases.

Physical therapy targeting posterior tibial muscle strengthening, calf stretching (Achilles contracture commonly develops in flatfoot and worsens arch mechanics), and intrinsic foot muscle strengthening is essential. A short course of anti-inflammatory medication helps during acute flares.

Surgery is recommended when a genuine trial of orthotics and physical therapy over 3–6 months has failed to control symptoms and functional impairment continues or the deformity is progressing.

The procedure

What Is Posterior Tibial Tendon Reconstruction (Flatfoot Correction)?

Posterior tibial tendon reconstruction corrects adult acquired flatfoot caused by a failing posterior tibial tendon. Surgery combines a tendon transfer (flexor digitorum longus) to replace the weakened tendon with bone realignment procedures — typically a calcaneal osteotomy and sometimes lateral column lengthening — to restore the foot's arch and correct malalignment.

Adult acquired flatfoot deformity (also called posterior tibial tendon dysfunction, or PTTD) is a progressive condition in which the posterior tibial tendon — the primary dynamic support of the medial arch — weakens and fails over time. As the tendon fails, the medial arch collapses, the heel tilts outward (hindfoot valgus), and the forefoot abducts (points outward). Patients develop pain along the inner ankle and arch, swelling, and inability to perform a single-leg heel rise on the affected foot.

PTTD is staged by severity, and treatment is matched to stage:

Stage I: The tendon is painful and inflamed (tenosynovitis) but has not yet lengthened significantly. The arch is maintained. Treatment: non-operative with orthotics and physical therapy. Surgery rarely needed.

Stage II: The tendon has elongated, the hindfoot has become valgus, and the arch has partially collapsed — but the deformity is still flexible (the heel corrects when standing on tiptoe). This is the primary surgical candidate stage. Reconstruction aims to restore alignment in a flexible foot.

Stage III: The deformity is rigid — the heel no longer corrects on tiptoe, and fixed arthritic changes are present. Reconstruction alone is insufficient; subtalar or hindfoot fusion may be necessary.

Stage IV: The deformity has propagated to the ankle joint, causing valgus tilt of the talus in the mortise. Ankle involvement requires more complex treatment.

This page focuses primarily on Stage II surgical correction — the most common reconstructive scenario.

What Happens During Posterior Tibial Tendon Reconstruction?

Surgery is performed at an ambulatory surgery center under regional or general anesthesia. The reconstructive plan is determined pre-operatively based on deformity stage and imaging, and may include the following components:

Anesthesia and positioning: The patient is positioned supine with the operative leg elevated slightly. Pneumatic tourniquets may be applied. A popliteal nerve block is typically used.

Component 1 — FDL Tendon Transfer to Navicular: A medial incision behind and below the medial malleolus exposes the posterior tibial tendon (degenerated, elongated, or torn) and the flexor digitorum longus tendon (lying just posterior to it). The degenerated PTT is excised. The FDL is harvested from its course behind the medial malleolus and transferred to the navicular bone — the primary attachment of the posterior tibial tendon. A bone tunnel is created in the navicular and the FDL is secured with an interference screw. This replaces the dynamic arch support function of the failed PTT.

Component 2 — Medializing Calcaneal Osteotomy (MCO): A lateral incision over the calcaneus is made. A diagonal osteotomy cut is made through the posterior calcaneus. The posterior heel fragment is shifted medially by approximately 8–10 mm, correcting the hindfoot valgus. The osteotomy is fixed with one or two large screws. This is the most critical static correction — realigning the heel under the leg restores the mechanical axis.

Component 3 — Lateral Column Lengthening (LCL), when needed: For feet with significant forefoot abduction (the forefoot pointing outward), a bone graft is inserted into the calcaneus at the calcaneocuboid joint, lengthening the lateral column and correcting forefoot abduction. LCL is added to MCO when abduction is significant.

Component 4 — Spring Ligament Repair: The spring (plantar calcaneonavicular) ligament, which supports the talar head, is often stretched in PTTD. Direct repair or imbrication of this ligament reinforces the medial column.

Component 5 — Subtalar Fusion (Stage III): For rigid flatfoot deformity, the subtalar joint (between talus and calcaneus) is fused rather than osteotomized. This permanently corrects hindfoot position but eliminates subtalar motion.

Closure: Multiple incisions are closed in layers. A well-padded posterior splint holds the foot in corrected position.

Recovery timeline

Days 1–14 (Posterior splint, strictly non-weight-bearing)

Multiple incisions require non-weight-bearing to protect the calcaneal osteotomy and tendon transfer simultaneously. Leg elevation at all times when not ambulating on crutches or a knee scooter.

Weeks 2–6 (Boot, non-weight-bearing)

Sutures removed at 2 weeks. Transition to CAM boot, but non-weight-bearing continues while the calcaneal osteotomy heals. X-rays at 6 weeks assess osteotomy healing.

Weeks 6–10 (Progressive weight-bearing in boot)

X-ray confirmation of osteotomy healing allows introduction of weight-bearing in the boot. Physical therapy begins — edema control, passive range of motion, gentle strengthening.

Months 3–4 (Shoe with custom orthotics, active PT)

Boot discontinued; transition to custom orthotic in a wide supportive shoe. Active physical therapy — peroneal and tibialis strengthening, arch-activation exercises, proprioception training.

Months 6–12 (Progressive activity return)

Walking is comfortable. Return to low-impact exercise (cycling, swimming) at 4–5 months. Return to higher-impact activity at 6–9 months as strength and endurance are rebuilt.

Month 12+ (Final outcome)

The calcaneal osteotomy is fully healed. The FDL transfer has matured. Most patients achieve a single-leg heel rise and return to normal activity. Final shoe fitting for custom orthotics (worn long-term) occurs at 6–12 months.

This is one of the longer and more demanding recoveries in foot and ankle surgery. The combined non-weight-bearing period of 6–8 weeks — during which neither the calcaneal osteotomy nor the tendon transfer can be safely loaded — is the primary challenge. Patients must plan their home environment, transportation, and work situation before surgery. Knee scooters are strongly recommended over crutches for most patients.

Custom orthotics are worn long-term after reconstruction to maintain the corrected arch alignment. The FDL transfer replaces the PTT but does not have identical strength — the orthotic supplements dynamic arch support permanently.

Physical therapy through Maryland Orthopedic Specialists is coordinated closely with the surgical milestones of osteotomy healing and tendon transfer maturation. The PT progression for this procedure is distinctly different from simpler ankle procedures and requires an experienced therapist familiar with flatfoot reconstruction.

Frequently Asked Questions

What is the difference between Stage II and Stage III flatfoot, and why does it matter for surgery?
Stage II flatfoot is flexible — the heel can still be passively corrected, and no fixed arthritic changes are present. This is surgically correctable with tendon transfer and osteotomy. Stage III flatfoot is rigid — the heel cannot be corrected and joint arthritis is established. Stage III requires fusion of one or more hindfoot joints, which eliminates motion to achieve a stable, pain-free foot. The staging drives the surgical plan completely, which is why pre-operative examination and imaging are so important.
Will I lose the ability to curl my toes after the FDL tendon transfer?
The FDL tendon bends the lesser toes, so harvesting it reduces strength in this action. In practice, most patients do not notice meaningful functional loss of toe curling because the FHL (flexor hallucis longus) maintains great toe flexion, and the intrinsic foot muscles contribute significantly to lesser toe function. Long-term studies show very low rates of clinically significant lesser toe dysfunction after FDL transfer.
Will I need to wear orthotics permanently after reconstruction?
Yes, in most cases. Custom orthotics are recommended long-term after flatfoot reconstruction to support the repaired arch and maintain alignment. The goal of surgery is to correct the bony structure so that orthotics can do their job efficiently — without the correction, no orthotic can adequately substitute for the absent arch support.
Why is recovery so long compared to other ankle surgeries?
The calcaneal osteotomy must heal before load-bearing through the hindfoot is safe. Bone healing requires 6–8 weeks of protected non-weight-bearing. Simultaneously, the FDL tendon transfer must begin to integrate at its navicular attachment. These two healing processes, running in parallel, determine the minimum non-weight-bearing period. Rushing weight-bearing risks osteotomy displacement and tendon transfer pullout.
Is there any alternative to surgery for Stage II flatfoot?
Custom orthotics and physical therapy can reduce symptoms significantly in many Stage II patients. An Arizona-style ankle-foot orthosis provides more aggressive support. However, orthotics do not halt tendon degeneration or prevent deformity progression. Patients who choose non-operative management for Stage II PTTD require monitoring for progression to Stage III, at which point surgical options become more limited.

Related conditions

Last reviewed May 20, 2026

References

  1. Myerson MS. Adult acquired flatfoot deformity: treatment of dysfunction of the posterior tibial tendon. Journal of Bone and Joint Surgery (American). 1996;78(5):780–792. doi:10.2106/00004623-199605000-00022. PMID: 40349380.
  2. Guyton GP, Jeng C, Krieger LE, Mann RA. Flexor digitorum longus transfer and medial displacement calcaneal osteotomy for posterior tibial tendon dysfunction: a middle-term clinical follow-up. Foot & Ankle International. 2001;22(8):627–632. doi:10.1177/107110070102200804. PMID: 11527022.